IMMUNE OSby Allerim

cytokine

IL-17

Barrier defense cytokine that recruits neutrophils and amplifies epithelial antimicrobial programs

TH17barrierneutrophilsautoimmunity

Review layer

Last reviewed 2026-05-17

conceptualeducational

Systems teaching draft. Content is structured for education and graph expansion, with formal source tagging ready for the next review pass.

State signature

Systems profile

Inflammation86
Tolerance45
Metabolism54
Tissue88
Neuroimmune38
Chronicity48

Cascade viewer

Source → signal → tissue behavior

Sources

T cell subsets
ILC3
gamma delta T cells

Signal

IL-17

Targets and effects

epithelium
fibroblasts
endothelium
CXCL8 induction
antimicrobial peptides
related: IL-6related: IL-13related: TGF-betarelated: GM-CSFrelated: CXCL8 / IL-8

Graph neighborhood

Direct relationships

Full graph

Barrier chemokine programs recruit neutrophils

IL-17associated withTH17 Cell

IL-17 is the signature TH17 effector signal

IL-17 and other inflammatory cytokines can amplify osteoclast-supporting osteoimmune signaling

IL-17 is the signature barrier recruitment signal for fungal and extracellular defense contexts

Cytokine ecology

Signal Role

IL-17 supports fungal and extracellular bacterial defense but becomes pathogenic when barrier or autoantigen loops persist.

Pathway

IL-17RA/RCACT1NF-kBC/EBP

Source Cells

T cell subsetsILC3gamma delta T cells

Target Cells

epitheliumfibroblastsendotheliummyeloid cells

Inflammatory Role

Induces chemokines and antimicrobial peptides that recruit and activate neutrophil-rich barrier responses.

Regulatory Role

Protects mucosal surfaces when microbiome and epithelial context are balanced.

Tissue Effects

CXCL8 inductionantimicrobial peptidesepithelial activationbarrier remodeling

Metabolic Effects

tracks with hypoxic and glycolytic inflammatory niches

Nervous System Interactions

can contribute to pain and neuroimmune inflammation in chronic barrier disease

Related Loops

IL-6 + TGF-beta -> TH17IL-17 -> CXCL8 -> neutrophil recruitment