IMMUNE OSby Allerim

cytokine

TSLP

Epithelial alarmin that primes type 2 immunity through dendritic cells, mast cells, and ILC2s

alarminepitheliumTH2allergy

Review layer

Last reviewed 2026-05-17

well-supportedclinical context required

Alarmin pages emphasize epithelial and stromal alarm signals as threshold-lowering inputs rather than isolated cytokine facts.

3 review sources

State signature

Systems profile

Inflammation86
Tolerance45
Metabolism54
Tissue88
Neuroimmune80
Chronicity48

Cascade viewer

Source → signal → tissue behavior

Sources

epithelium
keratinocytes
airway epithelial cells

Signal

TSLP

Targets and effects

dendritic cells
mast cells
ILC2
barrier alarm
itch biology
related: IL-4related: IL-33related: IL-5related: IL-13

Graph neighborhood

Direct relationships

Full graph

Epithelial alarmin primes dendritic cells toward TH2 instruction

TSLPactivatesMast Cell

Barrier stress signal that amplifies mast cell-rich allergic ecology

Keratinocyte alarmin output during itch and barrier stress

Cytokine ecology

Signal Role

TSLP is produced by stressed epithelium and pushes tissue immunity toward type 2 sensitization and allergic inflammation.

Pathway

TSLPRIL-7RαSTAT5

Source Cells

epitheliumkeratinocytesairway epithelial cells

Target Cells

dendritic cellsmast cellsILC2T cell subsets

Inflammatory Role

Promotes allergic sensitization, dendritic cell TH2 instruction, mast cell activation, and ILC2 cytokine output.

Regulatory Role

Can contribute to barrier repair signaling when transient and proportional.

Tissue Effects

barrier alarmitch biologyairway type 2 inflammationskin inflammation

Metabolic Effects

indirectly supports type 2 remodeling and repair metabolism

Nervous System Interactions

strongly linked to itch and sensory-neuroimmune barrier loops

Related Loops

epithelial stress -> TSLP -> DC TH2 primingTSLP -> ILC2/mast cell -> IL-5/IL-13