IMMUNE OSby Allerim

cytokine

IL-1beta

Inflammasome-linked alarm cytokine for fever, pain, neutrophil recruitment, and metabolic inflammation

inflammasomeNLRP3feverpain

Review layer

Last reviewed 2026-05-17

well-supportedclinical context required

Use as a conceptual pattern for innate danger sensing, metabolic stress, and IL-1beta-rich inflammatory states.

3 review sources

State signature

Systems profile

Inflammation86
Tolerance45
Metabolism82
Tissue62
Neuroimmune80
Chronicity78

Cascade viewer

Source → signal → tissue behavior

Sources

macrophages
monocytes
microglia
dendritic cells

Signal

IL-1beta

Targets and effects

endothelium
hypothalamic circuits
T cells
fever
pain sensitization
related: IL-6

Graph neighborhood

Direct relationships

Full graph

CNS danger sensing and sickness signaling

Signature inflammasome output

IL-1beta links danger sensing, fever, endothelial activation, and neutrophil-rich inflammation

Activated monocytes can produce IL-1 family inflammatory signals during innate priming

Cytokine ecology

Signal Role

IL-1beta is generated through priming and inflammasome activation, making it a signature of danger sensing, cell stress, and innate inflammatory thresholds.

Pathway

IL-1R1MyD88NF-kBMAPK

Source Cells

macrophagesmonocytesmicrogliadendritic cells

Target Cells

endotheliumhypothalamic circuitsT cellsneutrophilsfibroblasts

Inflammatory Role

Drives fever, pain sensitivity, endothelial activation, neutrophil recruitment, and TH17-supportive inflammation.

Regulatory Role

Useful for rapid danger response when time-limited and locally resolved.

Tissue Effects

feverpain sensitizationbarrier inflammationneutrophil recruitment

Metabolic Effects

insulin resistance contextmitochondrial stress couplingglycolytic inflammatory priming

Nervous System Interactions

fever circuitrysickness behaviormicroglial priming

Related Loops

danger signal -> inflammasome -> IL-1betaIL-1beta + IL-6 + TGF-beta -> TH17 conditions