IMMUNE OS ATLASby AllerimSign in

tissue

Adipose Immune Ecosystem

Metabolic immune niche where nutrient excess, hypoxia, macrophages, and fibrosis reshape systemic inflammation

metabolismmacrophagehypoxiainflammaging

Review layer

Last reviewed 2026-05-17

conceptualeducational

Systems teaching draft. Content is structured for education and graph expansion, with formal source tagging ready for the next review pass.

State signature

Systems profile

Inflammation86
Tolerance45
Metabolism82
Tissue88
Neuroimmune80
Chronicity78

System effects

Primary mapped axes from existing Atlas data.

Interpretive

Tissue

High 88

Organ, barrier, stromal, vascular, or local niche behavior.

vascular-endocrine interfaceextracellular matrix remodelingadipokine signalingmacrophages

Inflammation

High 86

Inflammatory alarm, recruitment, mediator release, or tissue-damaging amplification.

TNF-alphaIL-6IL-1betaIL-10

Metabolism

High 82

Energy allocation, glycolysis, mitochondrial strain, lipid signaling, or nutrient-sensitive behavior.

lipid overloadhypoxiainsulin resistancemitochondrial stress

Tissue ecosystem

Spatial immune niche

niche

Adipose

Barrier

vascular-endocrine interfaceextracellular matrix remodelingadipokine signaling

Cells

macrophagesT cell subsetsmast cellseosinophils

Cytokines

TNF-alphaIL-6IL-1betaIL-10

Neuroimmune

sympathetic lipolysisstress hormone effectsinflammatory pain and fatigue loops

Graph neighborhood

Direct relationships

Full graph

Arrow shows upstream source toward receiving target.

Adipocyte stress recruits and activates tissue macrophages

Immune niche

Tissue Ecology

Adipose tissue links energy storage to immune tone through adipocyte stress, macrophage crown-like structures, hypoxia, lipid mediators, and insulin signaling.

Dominant immune cells

macrophagesT cell subsetsmast cellseosinophilsNK cells

Barrier systems

vascular-endocrine interfaceextracellular matrix remodelingadipokine signaling

Microbiome interactions

gut-derived endotoxin toneSCFA effects on energy and inflammationbile acid-metabolic signaling

Neuroimmune interactions

sympathetic lipolysisstress hormone effectsinflammatory pain and fatigue loops

Cytokine environment

Metabolic conditions

lipid overloadhypoxiainsulin resistancemitochondrial stress