IMMUNE OSby Allerim

pathway

Vascular Biology & Endothelial Health

Endothelium, glycocalyx, nitric oxide, leukocyte adhesion, permeability, coagulation, and immune-vascular signaling.

coreendothelial glycocalyxnitric oxidevascular inflammationleukocyte adhesion

Review layer

Last reviewed 2026-05-17

conceptualeducational

Systems teaching draft. Content is structured for education and graph expansion, with formal source tagging ready for the next review pass.

core

Systems Frame

This module treats the vascular system as an active signaling organ. Cytokines, oxidative stress, ApoB retention, endothelial activation, and thromboinflammatory cues shift vascular behavior over time.

Core Concepts

endothelial glycocalyxnitric oxidevascular inflammationleukocyte adhesionthrombosismicrovascular dysfunctionarterial stiffness

Key Interactions

TNF-alpha and IL-1beta increase endothelial adhesion and permeability.

Oxidative stress impairs nitric oxide signaling and raises vascular tone.

ApoB retention and oxidized LDL recruit macrophages into plaque-like immune niches.

Coagulation and inflammation amplify each other during endothelial injury.

Scenario Examples

metabolic inflammation -> endothelial dysfunction

IL-1beta -> vascular activation

oxidized LDL -> foam cell biology

Build Next

endothelial injury graphApoB-to-foam-cell pathwaythromboinflammation visualizer